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Dexter Holland : ウィキペディア英語版 | Dexter Holland
Bryan Keith "Dexter" Holland (born December 29, 1965)〔(【引用サイトリンク】title=Dexter Holland Biography – Facts, Birthday, Life Story )〕〔(【引用サイトリンク】title=Dexter Holland – The Offspring Site )〕〔(【引用サイトリンク】title=Morning Minutes for Saturday, December 29 )〕 is an American musician and molecular biology graduate student, best known as the singer, rhythm guitarist, and primary songwriter for the punk rock band The Offspring. ==Education and scientific career== Holland attended Pacifica High School in Garden Grove, California, where he graduated as class valedictorian in 1984. During high school, Holland was the best student in mathematics in his year, and he found it "just as exciting as punk rock".〔(【引用サイトリンク】title=An Interview with Dexter Holland )〕 He then attended the University of Southern California, where he earned a BS in biology and an MS in molecular biology, and commenced a PhD in molecular biology. After the success of The Offspring, he suspended his candidature to focus on music. Holland had said in 1995 that, when he turns 40 years of age, he would rather be a professor at a university than play music.〔 As of 2013, Holland is a doctoral student at the Laboratory of Viral Oncology and Proteomics Research, Keck School of Medicine, where he is supervised by Professor Suraiya Rasheed.〔(Dexter Holland Discusses His Recently Published Paper 3/22/2013 – YouTube )〕 In March 2013, Holland and co-authors published a paper in ''PLoS One'' regarding microRNA in HIV genomes, titled "Identification of Human MicroRNA-Like Sequences Embedded within the Protein-Encoding Genes of the Human Immunodeficiency Virus".〔Bryan Holland, Jonathan Wong, Meng Li, Suraiya Rasheed. (2013.) ("Identification of Human MicroRNA-Like Sequences Embedded within the Protein-Encoding Genes of the Human Immunodeficiency Virus" ), ''PLoS One'', 8(3):1–10.〕 The original academic paper describes the use of computational molecular biological (''in silico'') approaches to identify microRNA-like sequences in HIV. These sequences are suggested to have evolved to self-regulate survival of the virus in the host by evading its immune responses and thus influence the persistence, replication, and pathogenicity of HIV.
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